Can contrast sensitivity functions in dyslexia be explained by inattention rather than a magnocellular deficit?
نویسندگان
چکیده
We examined whether data demonstrating contrast sensitivity losses in dyslexia that have been interpreted as evidence for loss of magnocellular visual function could be explained by inattention. Computer simulations of observers with poor concentration yielded inflated estimates of threshold that were a constant proportion of the true threshold across spatial frequencies. Data from many, but not all, studies supporting the magnocellular deficit theory are well described by these simulations, which predicted no interaction between observer group and spatial frequency. Some studies have reported significant interactions, but suffer from statistical deficiencies. This compromises some of the evidence for a magnocellular deficit in dyslexia derived from studies of threshold contrast sensitivity.
منابع مشابه
The magnocellular deficit theory of dyslexia: the evidence from contrast sensitivity
A number of authors have made the claim that dyslexia is the result of a deficit in the magnocellular part of the visual system. Most of the evidence cited in support of this claim is from contrast sensitivity studies. The present review surveys this evidence. The result of this survey shows that the support for the magnocellular deficit theory is equivocal. In the case of spatial contrast sens...
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The data bearing on the magnocellular or transient system deficit theory of dyslexia (Lovegrove et al., 1986, 1990; Livingstone et al., 1991) are highly conflicting. While some studies have produced results compatible with this theory (e.g. Lovegrove et al., 1982; Livingstone et al., 1991) other studies have yielded seemingly incompatible results (e.g. Smith et al., 1986; Victor et aZ., 1993; G...
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ورودعنوان ژورنال:
- Vision Research
دوره 41 شماره
صفحات -
تاریخ انتشار 2001